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		</div><p>People who suffer from sleep apnoea could benefit from the findings of a study on mice.</p>
<p>Central sleep apnoea occurs when there is a breakdown in the signals that regulate breathing during sleep, when oxygen levels are low.</p>
<p>Researchers at Edinburgh University used genetically modified mice to show that an enzyme called AMPK helps maintain a normal breathing pattern. In mice not producing AMPK, the appropriate signals are not sent and they fail to breathe faster when oxygen is low.</p>
<p>Central sleep apnoea commonly affects obese people and those with type 2 diabetes. It causes snoring, high blood pressure and poor memory, and sufferers are susceptible to falling asleep during the day.</p>
<p>It is also a problem at higher altitudes, where reduced oxygen levels can disrupt breathing even in fit and healthy people.</p>
<p>Professor Mark Evans from the university’s Centre for Integrative Physiology said: “Our findings identify exciting new avenues for the treatment of sleep disordered breathing, because drugs that mimic AMPK activation could restore normal breathing patterns in people suffering from this disease.</p>
<p>“Mice with AMPK deficiencies could also prove useful for helping us to identify such therapies.”</p>
<p>The study is published in the American Journal of Respiratory and Critical Care Medicine and was funded by the Wellcome Trust and the British Heart Foundation.</p>
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Quite a breakthrough.